Publicacións nas que colabora con Juan Parga Martín (20)

2024

  1. Non-HLA angiotensin-type-1 receptor autoantibodies mediate the long-term loss of grafted neurons in Parkinson’s disease models

    Stem Cell Research and Therapy, Vol. 15, Núm. 1

2023

  1. Combined cell-based therapy strategies for the treatment of Parkinson's disease: Focus on mesenchymal stromal cells

    Neural Regeneration Research, Vol. 18, Núm. 3, pp. 478-484

2021

  1. Dopamine regulates adult neurogenesis in the ventricular-subventricular zone via dopamine D3 angiotensin type 2 receptor interactions

    Stem Cells, Vol. 39, Núm. 12, pp. 1778-1794

  2. Dose-dependent effect of mesenchymal stromal cells co-grafted with dopaminergic neurons in a Parkinson's disease rat model

    Journal of Cellular and Molecular Medicine, Vol. 25, Núm. 20, pp. 9884-9889

  3. Nrf2 activation and downstream effects: Focus on parkinson’s disease and brain angiotensin

    Antioxidants, Vol. 10, Núm. 11

2019

  1. Interaction between Angiotensin Type 1, Type 2, and Mas Receptors to Regulate Adult Neurogenesis in the Brain Ventricular-Subventricular Zone

    Cells, Vol. 8, Núm. 12

2018

  1. Angiotensin II induces oxidative stress and upregulates neuroprotective signaling from the NRF2 and KLF9 pathway in dopaminergic cells

    Free Radical Biology and Medicine, Vol. 129, pp. 394-406

  2. Data on the effect of Angiotensin II and 6-hydroxydopamine on reactive oxygen species production, antioxidant gene expression and viability of different neuronal cell lines

    Data in Brief

  3. Prostaglandin EP2 Receptors Mediate Mesenchymal Stromal Cell-Neuroprotective Effects on Dopaminergic Neurons

    Molecular Neurobiology, Vol. 55, Núm. 6, pp. 4763-4776

2016

  1. Effects of rho kinase inhibitors on grafts of dopaminergic cell precursors in a rat model of Parkinson’s disease

    Stem Cells Translational Medicine, Vol. 5, Núm. 6, pp. 804-815

2012

  1. Mitochondrial ATP-sensitive potassium channels enhance angiotensin-induced oxidative damage and dopaminergic neuron degeneration. Relevance for aging-associated susceptibility to Parkinson's disease

    Age, Vol. 34, Núm. 4, pp. 863-880

2010

  1. Effect of inhibitors of NADPH oxidase complex and mitochondrial ATP-sensitive potassium channels on generation of dopaminergic neurons from neurospheres of mesencephalic precursors

    Developmental Dynamics, Vol. 239, Núm. 12, pp. 3247-3259

2009

  1. The mitochondrial ATP-sensitive potassium channel blocker 5-hydroxydecanoate inhibits toxicity of 6-hydroxydopamine on dopaminergic neurons

    Neurotoxicity Research, Vol. 15, Núm. 1, pp. 82-95

2008

  1. Brain angiotensin enhances dopaminergic cell death via microglial activation and NADPH-derived ROS

    Neurobiology of Disease, Vol. 31, Núm. 1, pp. 58-73

  2. Different effects of anti-sonic hedgehog antibodies and the hedgehog pathway inhibitor cyclopamine on generation of dopaminergic neurons from neurospheres of mesencephalic precursors

    Developmental Dynamics, Vol. 237, Núm. 4, pp. 909-917

2007

  1. Effects of GABA and GABA receptor inhibition on differentiation of mesencephalic precursors into dopaminergic neurons in vitro

    Developmental Neurobiology, Vol. 67, Núm. 12, pp. 1549-1559

  2. Mechanism of 6-hydroxydopamine neurotoxicity: The role of NADPH oxidase and microglial activation in 6-hydroxydopamine-induced degeneration of dopaminergic neurons

    Journal of Neurochemistry, Vol. 103, Núm. 1, pp. 145-156

  3. Serotonin decreases generation of dopaminergic neurons from mesencephalic precursors via serotonin type 7 and type 4 receptors

    Developmental Neurobiology, Vol. 67, Núm. 1, pp. 10-22

2005

  1. Expanded mesencephalic precursors develop into grafts of densely packed dopaminergic neurons that reinnervate the surrounding striatum and induce functional responses in the striatal neurons

    Synapse, Vol. 58, Núm. 1, pp. 13-22

2004

  1. Angiotensin II increases differentiation of dopaminergic neurons from mesencephalic precursors via angiotensin type 2 receptors

    European Journal of Neuroscience, Vol. 20, Núm. 6, pp. 1489-1498