Publicaciones (58) Publicaciones de María José López Armada

2022

  1. Involvement of Mitochondrial Dysfunction in the Inflammatory Response in Human Mesothelial Cells from Peritoneal Dialysis Effluent

    Antioxidants, Vol. 11, Núm. 11

  2. Mitochondrial Dysfunction and Oxidative Stress in Rheumatoid Arthritis

    Antioxidants, Vol. 11, Núm. 6

2021

  1. Autophagy Activation by Resveratrol Reduces Severity of Experimental Rheumatoid Arthritis

    Molecular Nutrition and Food Research, Vol. 65, Núm. 2

  2. Influencia de la ingesta de ácidos grasos omega-3, en la evolución de la inflamación presente en la artritis reumatoide.

    Nutrición clínica y dietética hospitalaria, Vol. 41, Núm. 3, pp. 111-122

  3. Mitochondrial dysfunction plays a relevant role in pathophysiology of peritoneal membrane damage induced by peritoneal dialysis

    Antioxidants, Vol. 10, Núm. 3, pp. 1-14

2019

  1. Role of mitochondrial dysfunction on rheumatic diseases

    Biochemical Pharmacology, Vol. 165, pp. 181-195

2017

  1. The mitochondrial inhibitor oligomycin induces an inflammatory response in the rat knee joint

    BMC Musculoskeletal Disorders, Vol. 18, Núm. 1

2016

  1. Resveratrol lowers synovial hyperplasia, inflammatory markers and oxidative damage in an acute antigen-induced arthritis model

    Rheumatology (United Kingdom), Vol. 55, Núm. 10, pp. 1889-1900

2015

  1. A pathogenetic role for endothelin-1 in peritoneal dialysis-associated fibrosis

    Journal of the American Society of Nephrology, Vol. 26, Núm. 1, pp. 173-182

2014

  1. Mitochondrial dysfunction promotes and aggravates the inflammatory response in normal human synoviocytes

    Rheumatology (United Kingdom), Vol. 53, Núm. 7, pp. 1332-1343

2013

  1. Mitochondrial dysfunction and the inflammatory response

    Mitochondrion, Vol. 13, Núm. 2, pp. 106-118

  2. Nitric oxide and the respiratory chain in synovial cells and chondrocytes

    Studies on Arthritis and Joint Disorders (Springer New York), pp. 49-66

2012

  1. Mitochondrial dysfunction increases inflammatory responsiveness to cytokines in normal human chondrocytes

    Arthritis and Rheumatism, Vol. 64, Núm. 9, pp. 2927-2936

2011

  1. Clinical significance of high levels of soluble tumour necrosis factor-α receptor-2 produced by alternative splicing in rheumatoid arthritis: A longitudinal prospective cohort study

    Rheumatology, Vol. 50, Núm. 4, pp. 721-728

2010

  1. Proteomic analysis by two-dimensional electrophoresis to identify the normal human chondrocyte proteome stimulated by tumor necrosis factor α and interleukin-1 β

    Arthritis and Rheumatism, Vol. 62, Núm. 3, pp. 802-814

2008

  1. Anti-apoptotic effect of transforming growth factor-β1 on human articular chondrocytes: role of protein phosphatase 2A

    Osteoarthritis and Cartilage, Vol. 16, Núm. 11, pp. 1370-1378

  2. Análisis proteómico bidimesional del condrocito humano normal bajo el efecto de IL-1β y TNF-α

    Proteómica: revista de la Sociedad Española de Proteómica, Núm. 1, pp. 83-83

  3. Differential effects of tumor necrosis factor-α and interleukin-1β on cell death in human articular chondrocytes

    Osteoarthritis and Cartilage, Vol. 16, Núm. 6, pp. 715-722

  4. Identificacion de proteínas que interaccionan con la subunidad catalítica de la fosfatasa 2A en condrocitos artrósicos y normales

    Proteómica: revista de la Sociedad Española de Proteómica, Núm. 1, pp. 87-87

  5. Mitochondrial dysfunction activates cyclooxygenase 2 expression in cultured normal human chondrocytes

    Arthritis and Rheumatism, Vol. 58, Núm. 8, pp. 2409-2419