Publicacións en colaboración con investigadores/as de Universidad Complutense de Madrid (21)

2023

  1. Safety and Efficacy of ApTOLL in Patients with Ischemic Stroke Undergoing Endovascular Treatment: A Phase 1/2 Randomized Clinical Trial

    JAMA Neurology, Vol. 80, Núm. 8, pp. 779-788

2022

  1. Cav-1 Protein Levels in Serum and Infarcted Brain Correlate with Hemorrhagic Volume in a Mouse Model of Thromboembolic Stroke, Independently of rt-PA Administration

    Molecular Neurobiology, Vol. 59, Núm. 2, pp. 1320-1332

2018

  1. Iron Overload Exacerbates the Risk of Hemorrhagic Transformation After tPA (Tissue-Type Plasminogen Activator) Administration in Thromboembolic Stroke Mice

    Stroke, Vol. 49, Núm. 9, pp. 2163-2172

2014

  1. Characterization of Gcf2/Lrrfip1 in experimental cerebral ischemia and its role as a modulator of Akt, mTOR and β-catenin signaling pathways

    Neuroscience, Vol. 268, pp. 48-65

  2. Oct-2 transcription factor binding activity and expression up-regulation in rat cerebral ischaemia is associated with a diminution of neuronal damage in vitro

    NeuroMolecular Medicine, Vol. 16, Núm. 2, pp. 332-349

  3. The high-mobility group I-Y transcription factor is involved in cerebral ischemia and modulates the expression of angiogenic proteins

    Neuroscience, Vol. 269, pp. 112-130

2013

  1. MiRNA expression is modulated over time after focal ischaemia: Up-regulation of miR-347 promotes neuronal apoptosis

    FEBS Journal, Vol. 280, Núm. 23, pp. 6233-6246

  2. ¿Qué ocurre con los artículos médicos remitidos en español que no son aceptados para publicación?

    Neurologia, Vol. 28, Núm. 4, pp. 205-211

2011

  1. A mouse model of hemorrhagic transformation by delayed tissue plasminogen activator administration after in situ thromboembolic stroke

    Stroke, Vol. 42, Núm. 1, pp. 196-203

  2. The natural tissue plasminogen activator inhibitor neuroserpin and acute ischaemic stroke outcome

    Thrombosis and Haemostasis, Vol. 105, Núm. 3, pp. 421-429

2007

  1. Increased body iron stores are associated with poor outcome after thrombolytic treatment in acute stroke

    Stroke, Vol. 38, Núm. 1, pp. 90-95

  2. Statin treatment withdrawal in ischemic stroke: A controlled randomized study

    Neurology, Vol. 69, Núm. 9, pp. 904-910

  3. The increase of circulating endothelial progenitor cells after acute ischemic stroke is associated with good outcome

    Stroke, Vol. 38, Núm. 10, pp. 2759-2764

2006

  1. A polymorphism in the EAAT2 promoter is associated with higher glutamate concentrations and higher frequency of progressing stroke

    Journal of Experimental Medicine, Vol. 203, Núm. 3, pp. 711-717

  2. High blood pressure and inflammation are associated with poor prognosis in lacunar infarctions

    Cerebrovascular Diseases, Vol. 22, Núm. 2-3, pp. 123-129

  3. Hyperthermia is a surrogate marker of inflammationmediated cause of brain damage in acute ischaemic stroke

    Journal of Internal Medicine, Vol. 260, Núm. 4, pp. 343-349

  4. Micro- and macroalbuminuria predict hemorrhagic transformation in acute ischemic stroke

    Neurology, Vol. 67, Núm. 7, pp. 1172-1177

  5. New-onset hypertension and inflammatory response/poor outcome in acute ischemic stroke

    Neurology, Vol. 67, Núm. 11, pp. 1973-1978

2005

  1. Increased plasma levels of 15-deoxy Δ prostaglandin J2 are associated with good outcome in acute atherothrombotic ischemic stroke

    Stroke, Vol. 36, Núm. 6, pp. 1189-1194

  2. The prediction of malignant cerebral infarction by molecular brain barrier disruption markers

    Stroke, Vol. 36, Núm. 9, pp. 1921-1926